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cyst fluid and surrounding inflammation), granular nodular (cyst retraction with decreasing

cyst fluid and surrounding inflammation), granular nodular (cyst retraction with decreasing

cyst fluid and surrounding inflammation), granular nodular (cyst retraction with decreasing

fluid-attenuated inversion recovery. Within 24 hours the patient developed hypotonia, worsening distal weakness (left greater than right), areflexia in all extremities, and diffuse pain. Blood testing for metabolic/endocrine abnormalities and serum inflammatory markers was unrevealing, aside from mildly elevated ESR. Autoimmune encephalitis and meningitis/encephalitis panels were

fluid-attenuated inversion recovery. Within 24 hours the patient developed hypotonia, worsening distal weakness (left greater than right), areflexia in all extremities, and diffuse pain. Blood testing for metabolic/endocrine abnormalities and serum inflammatory markers was unrevealing, aside from mildly elevated ESR. Autoimmune encephalitis and meningitis/encephalitis panels were

fluid-attenuated inversion recovery. Within 24 hours the patient developed hypotonia, worsening distal weakness (left greater than right), areflexia in all extremities, and diffuse pain. Blood testing for metabolic/endocrine abnormalities and serum inflammatory markers was unrevealing, aside from mildly elevated ESR. Autoimmune encephalitis and meningitis/encephalitis panels were

fluid filled cavity near the junction of the brainstem and upper cervical spine. Fig. 3.10 Blake pouch cyst

fluid filled cavity near the junction of the brainstem and upper cervical spine. Fig. 3.10 Blake pouch cyst

fluid filled cavity near the junction of the brainstem and upper cervical spine. Fig. 3.10 Blake pouch cyst

cysts. These abnormalities can be caused by chronic upper respiratory infection or allergic inflammation, which may not require treatment if asymptomatic (Fig. 1.3). However, the presence of air-fluid