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associated restricted diffusion. ADC, Apparent diffusion coefficient; DWI, diffusion weighted imaging. Pseudosubarachnoid hemorrhage: CT imaging appearance created by hypodense cerebral edema and/or ischemia with crowding of subarachnoid spaces. Vascular congestion due to compression and stasis may also contribute to extraaxial hyperdensity (Fig. 5.4). Panel A shows

aphasia, and seizures. Evaluation of vascular anatomy and perfusion is critical for diagnosis in acute ischemic or hemorrhagic stroke (HS) and other intracranial vasculopathies. Childhood vascular neurologic disease is distinct from that in adults, due to the varying risk factors from fetal life through adolescence. Hemorrhagic presentations

conduction velocities. Head CT and brain MRI showed diffuse cerebral white matter abnormality. The diagnosis was confirmed by skin biopsy for fibroblasts with enzymatic and molecular testing. Diagnostic Workup Brain MRI shows confluent and symmetric involvement of the periventricular and deep white matter, including splenium of corpus callosum, with

aphasia, and seizures. Evaluation of vascular anatomy and perfusion is critical for diagnosis in acute ischemic or hemorrhagic stroke (HS) and other intracranial vasculopathies. Childhood vascular neurologic disease is distinct from that in adults, due to the varying risk factors from fetal life through adolescence. Hemorrhagic presentations

aphasia. She also had features of dysautonomia, with unstable body temperature, labile blood pressure, and profuse sweating, which were noted to be aggravated by urinary retention or constipation. Diagnostic Workup CT of the brain showed that the patient had generalized cerebral atrophy. MRI showed patchy to confluent

associated restricted diffusion. ADC, Apparent diffusion coefficient; DWI, diffusion weighted imaging. Pseudosubarachnoid hemorrhage: CT imaging appearance created by hypodense cerebral edema and/or ischemia with crowding of subarachnoid spaces. Vascular congestion due to compression and stasis may also contribute to extraaxial hyperdensity (Fig. 5.4). Panel A shows

conduction velocities. Head CT and brain MRI showed diffuse cerebral white matter abnormality. The diagnosis was confirmed by skin biopsy for fibroblasts with enzymatic and molecular testing. Diagnostic Workup Brain MRI shows confluent and symmetric involvement of the periventricular and deep white matter, including splenium of corpus callosum, with

aphasia. She also had features of dysautonomia, with unstable body temperature, labile blood pressure, and profuse sweating, which were noted to be aggravated by urinary retention or constipation. Diagnostic Workup CT of the brain showed that the patient had generalized cerebral atrophy. MRI showed patchy to confluent

associated restricted diffusion. ADC, Apparent diffusion coefficient; DWI, diffusion weighted imaging. Pseudosubarachnoid hemorrhage: CT imaging appearance created by hypodense cerebral edema and/or ischemia with crowding of subarachnoid spaces. Vascular congestion due to compression and stasis may also contribute to extraaxial hyperdensity (Fig. 5.4). Panel A shows

aphasia, and seizures. Evaluation of vascular anatomy and perfusion is critical for diagnosis in acute ischemic or hemorrhagic stroke (HS) and other intracranial vasculopathies. Childhood vascular neurologic disease is distinct from that in adults, due to the varying risk factors from fetal life through adolescence. Hemorrhagic presentations