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infections (Fig. 5.2). Panel A shows an axial T1 M R I without contrast. The frontal and parietal lobes appear symmetric with visible cortical folds, lateral ventricles, and interhemispheric fissure. Brain structures are evenly defined, and sulci are uniformly spaced across both hemispheres. Panel B shows the corresponding axial

infections (Fig. 5.2). Panel A shows an axial T1 M R I without contrast. The frontal and parietal lobes appear symmetric with visible cortical folds, lateral ventricles, and interhemispheric fissure. Brain structures are evenly defined, and sulci are uniformly spaced across both hemispheres. Panel B shows the corresponding axial

infections (Fig. 5.2). Panel A shows an axial T1 M R I without contrast. The frontal and parietal lobes appear symmetric with visible cortical folds, lateral ventricles, and interhemispheric fissure. Brain structures are evenly defined, and sulci are uniformly spaced across both hemispheres. Panel B shows the corresponding axial

aneurysms, or brain tumors. He had no allergies to medications and his vaccinations were complete. Physical examination showed weight 55 kg (50th percentile), height 160 cm (50th percentile), head circumference 55 cm (75th percentile), blood pressure 110/70, pulse 88 bpm, and temperature

aneurysms, or brain tumors. He had no allergies to medications and his vaccinations were complete. Physical examination showed weight 55 kg (50th percentile), height 160 cm (50th percentile), head circumference 55 cm (75th percentile), blood pressure 110/70, pulse 88 bpm, and temperature

aneurysms, or brain tumors. He had no allergies to medications and his vaccinations were complete. Physical examination showed weight 55 kg (50th percentile), height 160 cm (50th percentile), head circumference 55 cm (75th percentile), blood pressure 110/70, pulse 88 bpm, and temperature

bacterial illness, which is thought to be an autoimmune process with cross-reactivity of pathogenic antibodies. This is most commonly described with varicella or Epstein-Barr virus, characterized by rapid-onset ataxia during the acute or convalescent phase of disease. Lumbar puncture usually reveals mild pleocytosis. Other etiologies

bacterial illness, which is thought to be an autoimmune process with cross-reactivity of pathogenic antibodies. This is most commonly described with varicella or Epstein-Barr virus, characterized by rapid-onset ataxia during the acute or convalescent phase of disease. Lumbar puncture usually reveals mild pleocytosis. Other etiologies

bacterial illness, which is thought to be an autoimmune process with cross-reactivity of pathogenic antibodies. This is most commonly described with varicella or Epstein-Barr virus, characterized by rapid-onset ataxia during the acute or convalescent phase of disease. Lumbar puncture usually reveals mild pleocytosis. Other etiologies

Aneurysmal subarachnoid hemorrhage, 39f Anisocoria, 319, 321 causes of, 319 Antecedent infections, 132 Antenatal infections