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infections (UTIs) than males.92 UTI is diagnosed when there is greater than 105 colony-forming units/mL in a urine culture. Notably, UTI is a separate entity from asymptomatic

asymptomatic bacteriuria a risk factor for prosthetic joint infection? Clin Infect Dis 1, 2014;59: 41-47. 17. Glynn

infection (PJI) following THA, so an undetectable viral load and a CD4+ count greater than 200 is recommended to optimize outcomes.86,87 In patients who report chronic corticosteroid use, it is recommended to taper these medications prior to the procedure and discuss considerations for a stress dose.88

infection. Sagittal T1 shows mild global volume loss and ex vacuo dilation of the prepontine cistern and fourth ventricle (arrows). (B) Axial T2 shows patchy edema in the bifrontal subcortical white matter (arrows), with crowding of sulci and basal cisterns. (C) Sagittal T1 with severe cerebral edema

infection (e.g., encephalitis, meningitis, sepsis), toxic ingestion, possible nonaccidental injury, and inborn errors of metabolism (IEMs). Imaging Differential Diagnoses In terms of IEMs, bilateral basal ganglia T2 hyperintensity raises the possibility of a primary mitochondrial disorder, pyruvate dehydrogenase complex deficiency, biotin-thiamine-responsive basal ganglia disease

infection. Sagittal T1 shows mild global volume loss and ex vacuo dilation of the prepontine cistern and fourth ventricle (arrows). (B) Axial T2 shows patchy edema in the bifrontal subcortical white matter (arrows), with crowding of sulci and basal cisterns. (C) Sagittal T1 with severe cerebral edema

infection (e.g., encephalitis, meningitis, sepsis), toxic ingestion, possible nonaccidental injury, and inborn errors of metabolism (IEMs). Imaging Differential Diagnoses In terms of IEMs, bilateral basal ganglia T2 hyperintensity raises the possibility of a primary mitochondrial disorder, pyruvate dehydrogenase complex deficiency, biotin-thiamine-responsive basal ganglia disease

infection (e.g., encephalitis, meningitis, sepsis), toxic ingestion, possible nonaccidental injury, and inborn errors of metabolism (IEMs). Imaging Differential Diagnoses In terms of IEMs, bilateral basal ganglia T2 hyperintensity raises the possibility of a primary mitochondrial disorder, pyruvate dehydrogenase complex deficiency, biotin-thiamine-responsive basal ganglia disease

infection. Sagittal T1 shows mild global volume loss and ex vacuo dilation of the prepontine cistern and fourth ventricle (arrows). (B) Axial T2 shows patchy edema in the bifrontal subcortical white matter (arrows), with crowding of sulci and basal cisterns. (C) Sagittal T1 with severe cerebral edema

infection is usually asymptomatic or mild in children with mainly respiratory symptoms. However,Page 213Page