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antibody syndrome, mixed connective tissue disease, sarcoidosis). Infectious causes of myelitis include EBV, CMV, poliovirus, herpes simplex virus (HSV), HIV, human T-lymphotropic virus type 1 (HTLV-1), Lyme, syphilis, tuberculosis, and schistosomiasis. As the patient’s course progressed, he developed areflexia more suggestive of lower motor

antibody syndrome, mixed connective tissue disease, sarcoidosis). Infectious causes of myelitis include EBV, CMV, poliovirus, herpes simplex virus (HSV), HIV, human T-lymphotropic virus type 1 (HTLV-1), Lyme, syphilis, tuberculosis, and schistosomiasis. As the patient’s course progressed, he developed areflexia more suggestive of lower motor

antibody syndrome, mixed connective tissue disease, sarcoidosis). Infectious causes of myelitis include EBV, CMV, poliovirus, herpes simplex virus (HSV), HIV, human T-lymphotropic virus type 1 (HTLV-1), Lyme, syphilis, tuberculosis, and schistosomiasis. As the patient’s course progressed, he developed areflexia more suggestive of lower motor

specific antibodies, including LGI1, GABABR, AMPAR, CASPR2, Hu, Ma2, and GAD. Both limbic encephalitis

specific finding. NMOSD is often associated with AQP-4 antibodies, which bind water channels

specific antibodies, including LGI1, GABABR, AMPAR, CASPR2, Hu, Ma2, and GAD. Both limbic encephalitis

specific finding. NMOSD is often associated with AQP-4 antibodies, which bind water channels

specific antibodies, including LGI1, GABABR, AMPAR, CASPR2, Hu, Ma2, and GAD. Both limbic encephalitis

specific finding. NMOSD is often associated with AQP-4 antibodies, which bind water channels

antibody-mediated demyelination, immune cell infiltration, mass-like formation, granulomatous inflammation, T-cell–mediated cytotoxic injury, and vasculitic inflammation. Early identification of the underlying systemic etiology of neurological disease is essential, as unchecked inflammation can cause severe neurological disability or even death. Furthermore, proper diagnosis can facilitate