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variation (prepubertal age and pregnancy), pathological causes (adenomas, hyperplasia, hypophysitis). Bilateral subdural collections: abusive head trauma with hematomas of varying ages, bridging vein injury, retinal hemorrhages (Fig. 4.4). The set of two M R I labeled A and B. Panel A shows an axial

variation in signal intensity. Vertical grid lines divide the timeline. The right panel shows a seven second electroencephalography segment with labeled channels from F7 to O2. A red box encloses the waveforms between F4 and O2. Additional channels labeled E C G and O S A T appear below, aligned

variation (prepubertal age and pregnancy), pathological causes (adenomas, hyperplasia, hypophysitis). Bilateral subdural collections: abusive head trauma with hematomas of varying ages, bridging vein injury, retinal hemorrhages (Fig. 4.4). The set of two M R I labeled A and B. Panel A shows an axial

variation in signal intensity. Vertical grid lines divide the timeline. The right panel shows a seven second electroencephalography segment with labeled channels from F7 to O2. A red box encloses the waveforms between F4 and O2. Additional channels labeled E C G and O S A T appear below, aligned

variation (prepubertal age and pregnancy), pathological causes (adenomas, hyperplasia, hypophysitis). Bilateral subdural collections: abusive head trauma with hematomas of varying ages, bridging vein injury, retinal hemorrhages (Fig. 4.4). The set of two M R I labeled A and B. Panel A shows an axial

variation in signal intensity. Vertical grid lines divide the timeline. The right panel shows a seven second electroencephalography segment with labeled channels from F7 to O2. A red box encloses the waveforms between F4 and O2. Additional channels labeled E C G and O S A T appear below, aligned