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behavioral problems, without fever or seizures at this time. Diagnostic Workup Brain MRI with contrast showed diffusion restriction and T2/fluid-attenuated inversion recovery (FLAIR) hyperintense signal in the bilateral thalami, insulae, insular and perirolandic cortex, and white matter (Fig. 37.1). There was no associated contrast enhancement or susceptibility

behavioral problems, without fever or seizures at this time. Diagnostic Workup Brain MRI with contrast showed diffusion restriction and T2/fluid-attenuated inversion recovery (FLAIR) hyperintense signal in the bilateral thalami, insulae, insular and perirolandic cortex, and white matter (Fig. 37.1). There was no associated contrast enhancement or susceptibility

behavioral problems, without fever or seizures at this time. Diagnostic Workup Brain MRI with contrast showed diffusion restriction and T2/fluid-attenuated inversion recovery (FLAIR) hyperintense signal in the bilateral thalami, insulae, insular and perirolandic cortex, and white matter (Fig. 37.1). There was no associated contrast enhancement or susceptibility

behavioral) over a time range of 6 months to 3 years. Many patients can manifest symptoms of adrenocortical insufficiency at the onset of symptoms, which should alert the physician to order laboratory testing and make a differential diagnosis. Of course, we also have to consider other differential

behavioral) over a time range of 6 months to 3 years. Many patients can manifest symptoms of adrenocortical insufficiency at the onset of symptoms, which should alert the physician to order laboratory testing and make a differential diagnosis. Of course, we also have to consider other differential

behavioral) over a time range of 6 months to 3 years. Many patients can manifest symptoms of adrenocortical insufficiency at the onset of symptoms, which should alert the physician to order laboratory testing and make a differential diagnosis. Of course, we also have to consider other differential