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pregnancy, with no family history. Birth examination showed a flat, alopecic, xanthomatous lesion in the left temporo-occipital scalp, suggestive of nevus sebaceous (Fig. 58.1). Neurological examination revealed asymmetry of spontaneous movements with preferential use of the left arm, as well as mild motor delay. In the first

pregnancy, with no family history. Birth examination showed a flat, alopecic, xanthomatous lesion in the left temporo-occipital scalp, suggestive of nevus sebaceous (Fig. 58.1). Neurological examination revealed asymmetry of spontaneous movements with preferential use of the left arm, as well as mild motor delay. In the first

pregnancy, with no family history. Birth examination showed a flat, alopecic, xanthomatous lesion in the left temporo-occipital scalp, suggestive of nevus sebaceous (Fig. 58.1). Neurological examination revealed asymmetry of spontaneous movements with preferential use of the left arm, as well as mild motor delay. In the first

loss with ex vacuo ventricular dilatation (arrow). (B–C) Axial SWI shows intraventricular hemorrhage staining the caudothalamic grooves (arrows) and choroid plexus. SWI, Susceptibility-weighted imaging. Final Diagnosis Unilateral spastic cerebral palsy (CP), due to periventricular venous infarction. Discussion Perinatal cerebrovascular accidents classically present with motor impairment, which

loss with ex vacuo ventricular dilatation (arrow). (B–C) Axial SWI shows intraventricular hemorrhage staining the caudothalamic grooves (arrows) and choroid plexus. SWI, Susceptibility-weighted imaging. Final Diagnosis Unilateral spastic cerebral palsy (CP), due to periventricular venous infarction. Discussion Perinatal cerebrovascular accidents classically present with motor impairment, which

loss with ex vacuo ventricular dilatation (arrow). (B–C) Axial SWI shows intraventricular hemorrhage staining the caudothalamic grooves (arrows) and choroid plexus. SWI, Susceptibility-weighted imaging. Final Diagnosis Unilateral spastic cerebral palsy (CP), due to periventricular venous infarction. Discussion Perinatal cerebrovascular accidents classically present with motor impairment, which

pregnancy give birth to affected infants. The incidence of neonates with CMV-specific symptoms at birth is reported at 10% to 15%. Of these, 40% to 60% develop permanent sequelae, particularlyPage 90Page 91sensorineural hearing loss followed by cognitive impairment, retinitis, and CP. Imaging findings depend on the developmental timing

pregnancy give birth to affected infants. The incidence of neonates with CMV-specific symptoms at birth is reported at 10% to 15%. Of these, 40% to 60% develop permanent sequelae, particularlyPage 90Page 91sensorineural hearing loss followed by cognitive impairment, retinitis, and CP. Imaging findings depend on the developmental timing

pregnancy give birth to affected infants. The incidence of neonates with CMV-specific symptoms at birth is reported at 10% to 15%. Of these, 40% to 60% develop permanent sequelae, particularlyPage 90Page 91sensorineural hearing loss followed by cognitive impairment, retinitis, and CP. Imaging findings depend on the developmental timing

pregnancy and birth history were unremarkable, and there was no family history of neurological disorders or other diseases. There was no parental consanguinity. Prior to the onset of symptoms, his school performance was normal. After the symptoms appeared, he began experiencing difficulties with his writing. The boy’s weight