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component and overall facilitated diffusion. Ependymomas arise from the ependymal cells of the fourth

component and overall facilitated diffusion. Ependymomas arise from the ependymal cells of the fourth

component and overall facilitated diffusion. Ependymomas arise from the ependymal cells of the fourth

component with small superior solid nodule (arrow). (D) Postcontrast coronal T1 shows peripheral cyst wall and solid nodular enhancement (arrows). Clinical Differential Diagnoses Common endocrine presentations of hypothalamic and pituitary disorders are central (vasopressin deficient) diabetes insipidus, hypopituitarism, precocious puberty, delayed/early puberty, and amenorrhea. In a patient

component with small superior solid nodule (arrow). (D) Postcontrast coronal T1 shows peripheral cyst wall and solid nodular enhancement (arrows). Clinical Differential Diagnoses Common endocrine presentations of hypothalamic and pituitary disorders are central (vasopressin deficient) diabetes insipidus, hypopituitarism, precocious puberty, delayed/early puberty, and amenorrhea. In a patient

component with small superior solid nodule (arrow). (D) Postcontrast coronal T1 shows peripheral cyst wall and solid nodular enhancement (arrows). Clinical Differential Diagnoses Common endocrine presentations of hypothalamic and pituitary disorders are central (vasopressin deficient) diabetes insipidus, hypopituitarism, precocious puberty, delayed/early puberty, and amenorrhea. In a patient

component in the retrobulbar space with encasement of the optic nerve, lateral rectus, and superior rectus-levator complex. (B) Axial T2 shows extrascleral invasion through the lateral globe (arrow), remodeling the lateral orbital wall. (C) Axial T1 postcontrast demonstrates avid heterogeneous tumor enhancement with overlying periorbital inflammation

component in the retrobulbar space with encasement of the optic nerve, lateral rectus, and superior rectus-levator complex. (B) Axial T2 shows extrascleral invasion through the lateral globe (arrow), remodeling the lateral orbital wall. (C) Axial T1 postcontrast demonstrates avid heterogeneous tumor enhancement with overlying periorbital inflammation

component in the retrobulbar space with encasement of the optic nerve, lateral rectus, and superior rectus-levator complex. (B) Axial T2 shows extrascleral invasion through the lateral globe (arrow), remodeling the lateral orbital wall. (C) Axial T1 postcontrast demonstrates avid heterogeneous tumor enhancement with overlying periorbital inflammation

cells with mucoid matrix. Immunohistochemistry showed positive results for synaptophysin and glial fibrillary acidic protein (GFAP) (in astrocytes) and negative neurofilament, CD34, and p53. Proliferative index (Ki-67 Ventana, clone 30-9) was 2% to 3%. Clinical Differential Diagnoses Focal epilepsies in children are often