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transplant candidates. The classic features are diffuse white matter abnormality, initially involving the deep white matter (usually posteriorly) with perivascular sparing. Fifteen percent of patients show a reverse pattern with predominant involvement of frontal white matter (Fig. 28.4). Isolated involvement of the corticospinal tracts can also occur

transplant candidates. The classic features are diffuse white matter abnormality, initially involving the deep white matter (usually posteriorly) with perivascular sparing. Fifteen percent of patients show a reverse pattern with predominant involvement of frontal white matter (Fig. 28.4). Isolated involvement of the corticospinal tracts can also occur

transplant candidates. The classic features are diffuse white matter abnormality, initially involving the deep white matter (usually posteriorly) with perivascular sparing. Fifteen percent of patients show a reverse pattern with predominant involvement of frontal white matter (Fig. 28.4). Isolated involvement of the corticospinal tracts can also occur

transplant occurred before or after the TKA.88 For dialysis patients, it appears that the mode of dialysis, hemodialysis (HD) versus peritoneal dialysis (PD), impacts infection risk. It has been shown that 1-year infection rates of dialysis-dependent patients undergoing THA or TKA are significantly lower

brain tissue was unrevealing. She stabilized on natalizumab for 3 years but subsequently progressed both clinically and radiologically. CSF phenotyping and soluble biomarker analysis revealed activation of her innate immune system. PRF1 gene sequencing identified a pathogenic mutation compatible with

brain tissue was unrevealing. She stabilized on natalizumab for 3 years but subsequently progressed both clinically and radiologically. CSF phenotyping and soluble biomarker analysis revealed activation of her innate immune system. PRF1 gene sequencing identified a pathogenic mutation compatible with

brain tissue was unrevealing. She stabilized on natalizumab for 3 years but subsequently progressed both clinically and radiologically. CSF phenotyping and soluble biomarker analysis revealed activation of her innate immune system. PRF1 gene sequencing identified a pathogenic mutation compatible with